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Future GLP-2 trials in IBD should stratify patients by disease phenotype (mucosal UC, inflammatory CD, stricturing CD) and incorporate serial cross-sectional imaging to monitor for stricture progression, given the mechanistic link between GLP-2R signaling through subepithelial myofibroblasts and intestinal fibrogenesis
Recombinant mouse OB protein: evidence for a peripheral signal linking adiposity and central neural networks
Three of the studies did not detect the products of oxidative metabolism following therapeutic dosing in children [44, 46, 48]
Should blood pressure control be the reason to start a GLP-1
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